Ditching the Dark Side of Dopamine: Avoiding Quick Fixes and Unhealthy Habits

Written by: Owen Tang

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Healf Journal

In today’s fast-paced world, many people turn to unhealthy “quick fix” sources of dopamine. Examples include substance abuse, junk food, chronic mobile device usage, and risky behaviour. These activities exploit the brain’s natural reward system, providing instant gratification through rapid dopamine release. While these “quick fixes” initially provide pleasure and satisfaction, they can have negative long-term consequences on the brain’s dopamine pathways, leading to reduced motivation, tolerance, and dependency. 

The science behind “quick fixes”

Fundamentally, the mechanics behind “quick fixes” and their effects on the brain’s dopamine pathways are similar: most “quick fix” activities trigger the rapid release of dopamine in the brain’s reward pathway, inducing feelings of pleasure and reinforcing the desire to repeat the action. 


Gradually, repetition causes changes in the brain: the brain adapts, reducing dopamine receptor sensitivity. Desensitised dopamine receptors result in tolerance, causing individuals to attempt “quick fixes” more frequently to achieve the same dopamine-releasing effect. 


With time, the brain’s natural ability to produce and/or regulate dopamine diminishes, leading to lesser self-motivation and poor mental well-being. The brain relies on “quick fixes” for dopamine, and stoppages may lead to withdrawals, potentially resulting in anxiety, irritability, and anhedonia. 


Afflicted individuals may attempt to self-medicate by continuing the unhealthy behaviour, temporarily boosting dopamine levels but eventually leading to dependency.

Sources of 'unhealthy' dopamine:

Substance abuse  


When experimenting with substances of abuse (alcohol, nicotine, opioids, stimulants, etc), most experience feelings of euphoria. Substance use causes this by triggering rapid dopamine release in the brain’s reward pathway, reinforcing the desire to use the substance repeatedly.

However, repeated use causes changes in the brain: the brain adapts, reducing dopamine receptor sensitivity and leading to tolerance. Individuals need to increase their substance usage to achieve the same effect. 


Eventually, the brain’s natural dopamine production diminishes, leading to low motivation and poor mental health. The brain has become reliant on substances for dopamine.


Chronic substance use may cause lasting changes to the brain’s dopamine pathways, making it difficult for individuals to derive pleasure from everyday activities, perpetuating the cycle of addiction since dopamine stimulation and normalcy are now achievable mainly through substance use. 


Overeating & Junk Food 


Like substance use, consuming foods high in sugar, fat, and salt affects the brain’s dopamine receptors. Eating such food triggers dopamine release in the nucleus accumbens, creating feelings of satisfaction and reinforcing the desire to eat more of it. 


Frequent junk food consumption leads to desensitisation of the brain’s dopamine receptors. As individuals attempt to stimulate dopamine release, this desensitisation may result in intense cravings and compulsive eating. 


The brain may eventually become less responsive to non-food rewards, making ordinarily pleasurable activities feel less rewarding. Long-term overconsumption of junk food may also alter the brain’s reward circuitry, making it harder to feel satisfied with smaller, healthier food portions. This alteration further increases the difficulty of switching from a highly processed diet to a healthy one, which may eventually lead to wider health issues. 


Digital Addiction 


The constant stimulation caused by digital platforms provides instant gratification through a continual influx of new content and arbitrary measures such as “likes" and "shares”. These trigger rapid dopamine release, causing pleasure and reinforcing the desire to continue engaging with online platforms. Many platforms also utilise variable reward schedules to surprise users and heighten dopamine responses, creating excitement similar to that of gambling, thereby further encouraging compulsive behaviours.


However, the overstimulation caused by the constant stream of information could lead to dopamine receptor desensitisation. Digitally addicted individuals may end up compulsively “doom-scrolling” social media or playing video games excessively to experience the same pleasure, eventually building up a dopamine tolerance similar to that experienced by substance abusers. 


Besides “doom-scrolling”, addicted individuals may experience less enjoyment from real-world activities. Constantly engaging with fast-paced digital content may also shorten attention span and impair focus, affecting their ability to complete longer tasks. 


Digital dependency and efforts to reduce digital platform usage may result in dopamine withdrawal symptoms like irritability and restlessness, since the brain has grown accustomed to constant dopamine stimulation. 


Risky Behaviours  


When engaging in risky behaviours, the brain interprets the uncertainty and unpredictability as novelty. This triggers a dopamine surge in anticipation of a reward, resulting in excitement and enjoyment. 


However, repeated risky acts can lead to the desensitisation of dopamine receptors, which may result in an increased risk tolerance as individuals undertake greater risk for the same dopamine-driven euphoria. 


The high stress levels involved in risky behaviour may also interact with dopamine, resulting in an even stronger dopamine response. This intense, exhilarating feeling may further reinforce the desire to repeat such acts, further endangering the participant. 

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Strategies to avoid unhealthy dopamine boosts

Rather than relying on “quick fixes” to stimulate dopamine and feel rewarded, individuals should chase healthy, sustainable sources of dopamine instead.  EATing  protein-rich foods, fruits, and vegetables can help optimise natural dopamine production while engaging in regular  MOVEment through physical activity and exercise promotes dopamine release.


Limiting Screen Time 


Limiting screen time can also benefit dopamine production and regulation, better enabling individuals to experience pleasure from organic, offline activities. 


Reducing screen time reduces overstimulation, which allows dopamine receptors to recover. Digital activities cause frequent dopamine spikes with instant gratification and variable rewards. By reducing screen time, the brain experiences fewer dopamine spikes, allowing the brain’s dopamine receptors to normalise and regain sensitivity. The brain can then naturally regulate dopamine levels better, improving emotional resilience and well-being.


Less screen time also improves focus and attention span. Reducing screen time limits the constant influx of dopamine triggers derived from digital platforms, gradually making the brain less distracted. It can then better engage in tasks requiring sustained effort, enhancing cognitive function over time. 


Limiting device usage is also beneficial for sleep quality and improving dopamine regulation. Blue light emitted by screens disrupts sleep patterns, causing poor sleep. Poor sleep impacts dopamine regulation, resulting in unstable moods and decreased motivation. Individuals can improve their sleep quality and maintain balanced dopamine levels by limiting screen time.

While unhealthy dopamine boosts from “quick fixes” like substance abuse, overeating, digital addiction, and risky behaviour provide short-term pleasure, they can cause long-term damage to the brain’s ability to produce and regulate dopamine naturally. Over time, this leads to desensitisation, compulsive behaviour, and diminished ability to enjoy life. To break free from these cycles, individuals must focus on healthier, sustainable sources of dopamine, such as physical activity, balanced nutrition, and mindful screen time management. Adopting these strategies makes it possible to restore balance to the brain’s dopamine system, improving overall well-being and resilience.


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This article is for informational purposes only, even if and regardless of whether it features the advice of physicians and medical practitioners. This article is not, nor is it intended to be, a substitute for professional medical advice, diagnosis, or treatment and should never be relied upon for specific medical advice. The views expressed in this article are the views of the expert and do not necessarily represent the views of Healf